52 research outputs found

    Wide QRS Tachycardia with Atrioventricular Dissociation and an HV Interval of 60 msec

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    Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/73193/1/j.1540-8167.1997.tb00814.x.pd

    Radiofrequency Ablation of Idiopathic Left Anterior Fascicular Tachycardia

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    Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/71580/1/j.1540-8167.1995.tb00389.x.pd

    Relative Timing of Isolated Potentials During Postinfarction Ventricular Tachycardia and Sinus Rhythm

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    Background: In postinfarction patients, isolated potentials separated by an isoelectric segment from the ventricular electrogram indicate areas of block. Isolated potentials can be recorded during both sinus rhythm and ventricular tachycardia (VT). In an attempt to differentiate bystander pathways from critical sites within a reentry circuit, we compared the relative timing of isolated potentials during VT and sinus rhythm.Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/46323/1/10840_2004_Article_5265641.pd

    Mapping and Ablation of Frequent Post-Infarction Premature Ventricular Complexes

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    Mapping of Post-Infarction PVCs .  Introduction: Premature ventricular complexes (PVCs) occur frequently in patients with heart disease. The sites of origin of PVCs in patients with prior myocardial infarction and the response to catheter ablation have not been systematically assessed. Methods and Results: In 28 consecutive patients (24 men, age 60 ± 10, ejection fraction [EF] 0.37 ± 0.14) with remote myocardial infarction referred for catheter ablation of symptomatic refractory PVCs, the PVCs were mapped by activation mapping or pace mapping using an irrigated-tip catheter in conjunction with an electroanatomic mapping system. The site of origin (SOO) was classified as being within low-voltage (scar) tissue (amplitude ≀1.5 mV) or tissue with preserved voltage (>1.5 mV). The SOO was confined to endocardial scar tissue in 24/28 patients (86%). The SOO was outside of scar in 3 patients and could not be identified in 1 patient. At the SOO, local endocardial activation preceded the PVC by 46 ± 19 ms, and the electrogram amplitude during sinus rhythm was 0.48 ± 0.34 mV. The PVCs were effectively ablated in 25/28 patients (89%), resulting in a decrease in PVC burden on a 24-hour Holter monitor from 15.6 ± 12.3% to 2.4 ± 4.2% (P < 0.001). The SOO most often was confined to scar tissue located in the left ventricular septum and the papillary muscles. Conclusion: Similar to post-infarction ventricular tachycardia, PVCs after remote myocardial infarction most often originate within scar tissue. Catheter ablation of these PVCs has a high-success rate. (J Cardiovasc Electrophysiol, Vol. 21, pp. 1002-1008, September 2010)Peer Reviewedhttp://deepblue.lib.umich.edu/bitstream/2027.42/79266/1/j.1540-8167.2010.01771.x.pd
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